MKGC: a curated molecular knowledgebase for gastric cancer
 Browse All GCs By
  Summary of source.
Source Count
blood 4
cell line 365
exosomes in plasma 2
mice 62
plasma 2
PPLF 1
serum 6
tissue 259
  Browse for lncRNA
Official
Symbol		 type pmid method Sample
Type		 Region Sample
Number
GC		 Sample
Number
nonGC		 Normal
vs.
GC		 Early-stage
vs.
advanced-stage		 Hpylori Result Functional
Type		 Overall
Survival		 Disease-free
Survival		 AUC
ABHD11-AS1 lncRNA 24984296 qRT-PCR tissue China 75 75 Up - No Reports Compared with adjacent non-tumor tissues the expression level of ABHD11-AS1 in gastric cancer tissues was significantly increased (P = 0.027). The expression level was also significantly related with the differentiation (P = 0.022), Lauren histologic classification (P = 0.004) and carbohydrate antigen 19-9 (CA19-9) (P = 0.007), and the area under ROC curve was up to 0.613 diagnosis - - 0.613
AFAP1-AS1 lncRNA 28451917 qRT-PCR tissue China 20 20 Up - No Reports AFAP1-AS1 was up-regulated in GC tissues and GC cells. - - - -
AFAP1-AS1 lncRNA 28451917 Cell proliferation assay; Colony formation assay; Cell apoptosis assay cell line - - - - - No Reports AFAP1-AS1 knockdown suppressed cell viability of SGC7901 transfected with si-AFAP1-AS1. The number of apoptotic SGC7901 cell transfected with si-AFAP1-AS1 was increased by 3.4-fold comparing to that of control. - - - -
AFAP1-AS1 lncRNA 28975981 qRT-PCR tissue China 91 91 Up Up No Reports AFAP1-AS1 expression was dramatically increased in GC tissues and cells, compared with noncancerous gastric tissues and control cells. The higher lncRNA AFAP1-AS1 expression was positively correlated with lymph node metastasis, TNM stage and worse overall survival (OS) time in GC patients. AFAP1-AS1 expression levels were independent prognostic factors of OS in GC patients. prognosis 0.001 - -
AFAP1-AS1 lncRNA 28975981 Cell proliferation assay; Invasion assay; cell line - - - - - No Reports Knockdown of lncRNA AFAP1-AS1 significantly inhibited the cell proliferation and cell cycle progression. Reduced lncRNA AFAP1-AS1 also inhibited the cell invasion ability via regulating cell epithelial-mesenchymal transition (EMT) phenomenon in GC. prognosis 0.001 - -
AGAP2-AS1 lncRNA 28209205 qRT-PCR tissue China 50 50 Up - No Reports AGAP2-AS1 was highly expressed in the GC tissues and cell lines, and patients with higher AGAP2-AS1 expression had a poorer prognosis and shorter overall survival. Higher AGAP2-AS1 level was associated with larger tumors, advanced pathological stage, and lymph node metastasis. prognosis 0.002 0.025 -
AGAP2-AS1 lncRNA 28209205 Cell proliferation assay; Invasion and migration assay; Cell apoptosis assay; Tumor formation assay cell line - - - - - No Reports Knockdown of AGAP2-AS1 significantly inhibited GC cell proliferation, migration, and invasion in vitro and tumor growth in vivo. prognosis 0.002 0.025 -
AGAP2-AS1 lncRNA 28209205 Luciferase reporter assay; ChIP assay cell line - - - - - No Reports SP1 could bind to all of these promoter regions of AGAP2-AS1 prognosis 0.002 0.025 -
AGAP2-AS1 lncRNA 28209205 RNA immunoprecipitation cell line - - - - - No Reports AGAP2-AS1 functions as an oncogenic lncRNA by interacting with LSD1 and EZH2 and suppressing CDKN1A (P21) and E-cadherin transcription. prognosis 0.002 0.025 -
AK023391 lncRNA 29282102 qRT-PCR; Microarray tissue China 5 5 Up - No Reports Expression of lncRNA AK023391 was significantly upregulated in gastric cancer samples and cell lines in comparison to adjacent normal tissues prognosis 0.0024 - 0.56
AK023391 lncRNA 29282102 FISH tissue China 77 77 Up - No Reports AK023391 expression was upregulated in GC and was mainly localized in the cytoplasm of the tissue cell, and was positively correlated with poor survival in patients with gastric cancer. The multivariate Cox regression model revealed that AK023391 expression acted as an independent prognostic factor for survival in patients with gastric cancer. prognosis 0.0024 - 0.56
AK023391 lncRNA 29282102 Cell viability assay; Invasion and migration assay; Colony formation assay; Flow cytometry cell line - - - - - No Reports Knockdown of AK023391 inhibits the proliferation, colony formation, DNA synthesis, migration, invasion of GC cells, and induces apoptosis and cell cycle arrest. prognosis 0.0024 - 0.56
AK023391 lncRNA 29282102 IHC; Western blot cell line - - - - - No Reports LncRNA AK023391 is involved in the regulation of the PI3K/Akt signaling pathway prognosis 0.0024 - 0.56
AK023391 lncRNA 29282102 Xenograft mouse models mice - - - - - No Reports Knockdown of lncRNAAK023391 inhibits tumor growth in vivo prognosis 0.0024 - 0.56
BANCR lncRNA 26054683 qRT-PCR tissue China 20 20 Up - No Reports The expression of BANCR was increased in gastric cancer tissues compared with paired adjacent normal tissues. prognosis < 0.001 - -
BANCR lncRNA 26054683 qRT-PCR tissue China 184 - - - No Reports High expression of BANCR was positively associated with clinical stage, tumor depth, lymph node metastasis and distant metastasis in gastric cancer patients. prognosis < 0.001 - -
BNC2-AS1 lncRNA 30549996 Cell proliferation assay; Cell invasion and migration assays; Cell colony forming assay; cell line - - - - - No Reports BNC2-AS1-knockdown significantly suppressed gastric cancer cell proliferation, migration and suppressed gastric cancer cell invasion - - - -
CASC15 lncRNA 29272000 qRT-PCR tissue China 60 42 Up Up No Reports Expression of lncRNA CASC15 in gastric cancer tissue was higher than normal gastric epithelium. Clinical data analysis showed that the expression level of CASC15 was correlated with tumor size and TNM stage in clinical patients. prognosis 0.013 - -
CASC15 lncRNA 29272000 Cell proliferation assay cell line - - - - - No Reports Overexpression of LncRNA CASC15 promoted the proliferation of MKN28 cells. prognosis 0.013 - -
CCAT1 lncRNA 28239816 qRT-PCR tissue China 240 240 Up - No Reports The expression of CARLo-5 in gastric cancer tissues was significantly higher than that of their matched adjacent normal tissues. High CARLo-5 expression was found to be closely correlated with advanced T stage, positive distant metastasis, lymph node involvement, and poor differentiation. Kaplan-Meier analysis demonstrated that gastric cancer patients with high CARLo-5 expression had poorer OS (p < 0.001) and RFS (p < 0.001). Finally, univariate and multivariate Cox analysis indicated that high CARLo-5 expression were independent predictors for both OS and RFS. prognosis < 0.001 - -
CCAT1 lncRNA 28535628 qRT-PCR tissue China 62 62 Up - No Reports The expressions of CCAT1 mRNA in GC tissues were significantly higher than in the normal tissues.The expression of CCAT1 varied significantly among patients with different TNM stage, depth of invasion, and lymph node metastasis. target - - -
CCAT1 lncRNA 28535628 Cell proliferation assay cell line - - - - - No Reports Down-regulation of CCAT1 in MGC-803 and SGC-7901 cells significantly inhibited the cell proliferation. target - - -
CCAT2 lncRNA 28248065 qRT-PCR tissue China 208 208 Up - No Reports lncRNA CCAT2 was upregulated in GC tissues (P=0.000), and positively correlated with TNM stage (P=0.029), lymphatic invasion (P=0.042) and nervous invasion (P=0.024) in GC patients. Furthermore, we also found that high expression of lncRNA CCAT2 was an unfavorable prognostic factor in GC patients. prognosis < 0.05 - -
CCAT2 lncRNA 28248065 Cell proliferation assay; Invasion and migration assays cell line - - - - - No Reports Silencing of lncRNA CCAT2 inhibits gastric cancer cell proliferation and invasion. prognosis < 0.05 - -
CCAT2 lncRNA 25755774 qRT-PCR tissue China 85 85 Up - No Reports Expression levels of lncRNA CCAT2 in gastric cancer tissues were significantly higher than those in adjacent non-tumor tissues. High lncRNA CCAT2 expression was observed to be closely correlated with higher incidence of lymph node metastasis and distance metastasis. Moreover, patients with high lncRNA CCAT2 expression had shorter overall survival and progression-free survival compared with the low lncRNA CCAT2 group. Multivariate analyses indicated that high lncRNA CCAT2 expression was an independent poor prognostic factor for gastric cancer patients. prognosis 0.003 - -
CCEPR lncRNA 29771412 qRT-PCR tissue China 77 77 Up - No Reports Expression of lncRNA CCHE1 in the tumor cell lines was increased (p<0.05).Bcl-2 protein expression increased, but Bax protein expression decreased in lncRNA CCHE1 upregulation group (p<0.05); the effect was reversed in lncRNA CCHE1 down-regulation group (p<0.05). target - - -
CCEPR lncRNA 29771412 Cell proliferation assay; Cell colony formation assay; Flow cytometry cell line - - - - - No Reports Down-regulated lncRNA CCHE1 decreased proliferative activity and colony formation capacity.Cell cycle was promot_x005fed in lncRNA CCHE1 up-regulation group. target - - -
CDKN2B-AS1 lncRNA 31242038 PCR tissue China 192 - - - No Reports Patients with higher ANRIL survived worse. Expressions of TET2 and ANRIL were negatively correlated in the patient samples. target/prognosis < 0.001 - -
CDKN2B-AS1 lncRNA 31242038 Apoptosis assay; Migration assay cell line - - - - - No Reports ANRIL suppresses apoptosis in gastric cancer cell, enhances metastasis target/prognosis < 0.001 - -
CDKN2B-AS1 lncRNA 31242038 Xenograft tumor mouse model cell line - - - - - No Reports ANRIL promotes tumor growth in vivo target/prognosis < 0.001 - -
CDKN2B-AS1 lncRNA 30156609 qRT-PCR tissue China 20 20 Up - No Reports ANRIL level was elevated in gastric cancer tissues and cell lines. Furthermore, ANRIL silence down-regulated BMI1 via up-regulating miR-99a. BMI1 silence down-regulated Bcl-2 and key kinases in the Notch and mTOR pathways and up-regulated p16 and cleaved caspases. target - - -
CDKN2B-AS1 lncRNA 30156609 Cell proliferation assay; Invasion and migration assays; Flow cytometry cell line - - - - - No Reports Knockdown of ANRIL suppressed cell viability, migration, and invasion, and increased apoptosis through up-regulating miR-99a. target - - -
CDKN2B-AS1 lncRNA 30156609 qRT-PCR; Cell transfection cell line - - - - - No Reports ANRIL silence_x005finduced up-regulation of miR-99a was significantly reversed by miR-99a inhibitor.ANRIL knockdown down-regulated the expression of BMI1 by modulating miR-99a in gastric cancer cells in vitro target - - -
CRNDE lncRNA 28490034 qRT-PCR tissue China 20 20 Up - No Reports CRNDE was highly expressed in GC cell lines and tissues; target - - -
CRNDE lncRNA 28490034 Cell proliferation assay cell line - - - - - No Reports Overexpression of CRNDE increased GC cell viability and promoted colony formation. Knockdown of CRNDE did not result in loss of expression-related effects on cell proliferation and colony formation. target - - -
CRNDE lncRNA 28490034 Luciferase reporter assay cell line - - - - - No Reports miR-145 target gene E2F3 was strongly expressed following CRNDE competitive molecular sponging of miR-145. target - - -
CRNDE lncRNA 29243780 qRT-PCR tissue China 118 118 Up - No Reports Expression of CRNDE was higher in GC tissues and cells compared with the normal gastric tissue and normal gastric cell lines. High expression of CRNDE was correlated with invasion depth, TNM stage and lymph node metastasis. Furthermore, high CRNDE expression was associated with shorter overall survival of GC patients. Multivariate analysis confirmed that high CRNDE expression was a significant independent predictor of poor survival in GC. prognosis 0.0066 - -
CRNDE lncRNA 29243780 Cell proliferation assay; Cell invasion and migration assays cell line - - - - - No Reports Knockdown of CRNDE inhibited cell proliferation, migration and invasion of GC. prognosis 0.0066 - -
CRNDE lncRNA 29243780 Western blot; cell transfection cell line - - - - - No Reports CRNDE exerted its oncogenic role by affecting PI3K/AKT signaling pathways. prognosis 0.0066 - -
DUSP5P1 lncRNA 30733230 Luciferase reporter assay; Western blot; qRT-PCR; ChIP-PCR cell line - - - - - No Reports DUSP5P1 is a direct downstream target of C8orf76 mechanism - - -
DUSP5P1 lncRNA 30733230 Cell proliferation assay; Cell colony formation assay; Invasion and migration assays; Flow cytometry cell line - - - - - No Reports DUSP5P1 promotes gastric cancer cell growth in vitro. mechanism - - -
DUSP5P1 lncRNA 30733230 Xenograft mice models mice - - - - - No Reports Significantly more proliferating cells and fewer apopto- tic cells were shown in DUSP5P1-transfected xenografts as compared with control group by Ki-67 and TUNEL assays. mechanism - - -
FLJ22763 lncRNA 30716442 qRT-PCR tissue China 5 5 Down - No Reports FLJ22763, a novel lncRNA, had significantly lower expression in GC tissues. target/prognosis 0.003 - -
FLJ22763 lncRNA 30716442 qRT-PCR tissue China 303 - - Down No Reports Decreased expression of FLJ22763 was positively correlated with a lower-level histological grade and the depth of invasion. Patients with high FLJ22763 expression levels had a higher five-year survival rate. target/prognosis 0.003 - -
FLJ22763 lncRNA 30716442 Cell proliferation assay; Invasion and migration assays cell line - - - - - No Reports LncRNA FLJ22763 inhibits cells proliferation, migration, and invasion target/prognosis 0.003 - -
FLJ22763 lncRNA 30716442 Xenograft mice models mice - - - - - No Reports LncRNA FLJ22763 inhibits tumorigenesis of GC cells in vivo target/prognosis 0.003 - -
FLJ22763 lncRNA 30716442 qRT-PCR; Cell tranfection; Western blot cell line - - - - - No Reports FLJ22763 was negatively associated with ACLY, regulating the mRNA and protein levels of ACLY target/prognosis 0.003 - -
GAS5 lncRNA 25959498 qRT-PCR tissue China 55 55 Down - No Reports lncRNA GAS5 had lower expression in stomach cancer tissues than the normal counterparts. p21 expression correlates with lncRNA GAS5 expression in stomach cancer tissues. target - - -
GAS5 lncRNA 25959498 Flow cytometry cell line - - - - - No Reports lncRNA GAS5 knock-down abolishes cell cycle arrest at the G1 phase target - - -
GAS5 lncRNA 25959498 RNA pull-down assay cell line - - - - - No Reports lncRNA GAS5 interacts with the transcriptional activator YBX1. target - - -
H19 lncRNA 28848149 qRT-PCR tissue China 34 34 Up - No Reports H19 was increased in gastric cancer cell lines and tissues. Overexpression of H19 promoted cell proliferation and inhibited cell apoptosis, whereas knockdown of H19 inhibited these effects. By further examining the underlying mechanism, we showed that H19/miR-675 axis inhibited expression of FADD. FADD downregulation subsequently inhibited the caspase cleavage cascades including caspase 8 and caspase 3. mechanism - - -
H19 lncRNA 28848149 Cell proliferation assay; Colony formation assay; Flow cytometry cell line - - - - - No Reports Overexpression of H19 and miR-675 promoted cell proliferation and inhibited cell apoptosis, whereas knockdown of H19 and miR-675 inhibited these effects. mechanism - - -
HOTAIR lncRNA 30810117 ISH tissue China 30 30 Up - No Reports HOTAIR expression of cancer tissues was significantly increased compared with that of non_x005ftumor tissues; lncRNA HOTAIR was negatively correlated with miR-454-3p expression in gastric cancer tissues. mechanism - - -
HOTAIR lncRNA 30810117 Cell proliferation assay; Flow cytometry cell line - - - - - No Reports HOTAIR knockdown had anti-tumor effects that suppressed gastric cancer cell proliferation by increasing cell apoptosis and keeping the cell cycle in G1 phase. mechanism - - -
KRT19P3 lncRNA 31409899 qRT-PCR tissue China 84 29 Down - No Reports KRT19P3 expression was significantly decreased in gastric cancer tissues. Lower expression of KRT19P3 was found to be associated with lager tumor size (≥ 5 cm), advanced TNM stage, Lauren's classification, and positive LNM. Patients with lower KRT19P3 expression had worse overall survival (OS) and disease-free sur_x005fvival (DFS) compared with those patients with higher KRT19P3 expression Univariate analysis demonstrated that patients with lower KRT19P3 expression tended to have a higher risk of death. target/prognosis 0.0016 0.0028 -
KRT19P3 lncRNA 31409899 Cell proliferation assay; Cell invasion and migration assays; cell line - - - - - No Reports KRT19P3 inhibited GC cell proliferation and induced apoptosis, suppressed GC cell migration and invasion in vitro. target/prognosis 0.0016 0.0028 -
KRT19P3 lncRNA 31409899 Xenograft mice models mice - - - - - No Reports KRT19P3 suppressed GC cell migration and invasion and tumor growth in vivo. target/prognosis 0.0016 0.0028 -
KRT19P3 lncRNA 31409899 RNA pull-down assay; RIP; Western blot cell line - - - - - No Reports KRT19P3 could bind COPS7A directly and might regulate COPS7A activity in GC target/prognosis 0.0016 0.0028 -
LINC01089 lncRNA 29717028 qRT-PCR tissue China 32 32 Up - No Reports lncRNA AK096174 was up-regulated and associated with tumorigenesis, tumor size, metastasis, and poor prognosis in GC. Our data showed that AK096174 was highly expressed in the GC tissues and cell lines (SGC-7901, AGS, BGC-823, MGC-803), and patients with higher AK096174 expression had a poorer prognosis and shorter overall survival (OS). AK096174 knockdown inhibited the proliferation, migration, and invasiveness in SGC-7901 and BGC-823 cells, whereas AK096174 overexpression had the promoting effects. Furthermore, mechanistic investigation showed that AK096174 positively correlated with the expression of WD repeat-containing protein 66 (WDR66) gene at the translational level. Knockdown of WRD66 attenuated the positive impact of AK096174 in GC cells target - - -
LINC01089 lncRNA 29717028 Cell proliferation assay; Cell invasion and migration assays; cell line - - - - - No Reports AK096174 knockdown inhibited the proliferation, migration, and invasiveness in SGC-7901 and BGC-823 cells, whereas AK096174 overexpression had the promoting effects. target - - -
LINC01234 lncRNA 29386218 qRT-PCR tissue China 50 50 Up - No Reports LINC01234 expression was significantly upregulated in gastric cancer tissues and was associated with larger tumor size, advanced TNM stage, lymph node metastasis, and shorter survival time. prognosis <0.001 <0.001 -
LINC01234 lncRNA 29386218 Cell proliferation assay; Invasion and migration assays; Flow cytometry cell line - - - - - No Reports promote cell proliferation and cell tomorigenesis, suppress apotosis and G1 arrest prognosis <0.001 <0.001 -
LINC01234 lncRNA 29386218 Xenograft mice models mice - - - - - No Reports knockdown of LINC01234-induced apoptosis and growth arrest in vitro and inhibited tumorigenesis in mouse xenografts. prognosis <0.001 <0.001 -
LINC01234 lncRNA 29386218 Immunoprecipitation assays cell line - - - - - No Reports LINC01234 binds directly to Ago2 prognosis <0.001 <0.001 -
LINC01234 lncRNA 29386218 Luciferase reporter assay cell line - - - - - No Reports miR-204-5p directly target LINC01234 prognosis <0.001 <0.001 -
LINC02859 lncRNA 31207155 qRT-PCR tissue China 90 90 Down - No Reports LOC101928316 was significantly downregulated in gastric cancer (GC) tissues specimen, GC cell lines, and associated with the GC patients tumor, node, and metastasis (TNM) stage and degree of differentiation. target - - -
LINC02859 lncRNA 31207155 Cell proliferation assay; Invasion and migration assay; Flow cytometry cell line - - - - - No Reports LOC101928316 overexpression can significantly inhibit GC cell migration, invasion, and proliferation. target - - -
LINC02859 lncRNA 31207155 Xenograft mouse models mice - - - - - No Reports LOC101928316 Overexpression inhibited gastric cancer cells tumorigenesis in vivo target - - -
LINC02859 lncRNA 31207155 Western blot cell line - - - - - No Reports lenti-LOC101928316-Overexpression stable infected can remarkably inhibit the expression level of PI3K, p-AKT, mTOR, and p-mTOR in GC cells target - - -
NMRAL2P pseudogene/lncRNA 32469171 qRT-PCR tissue China 10 10 Down - No Reports NMRAL2P was downregulated in tumor tissues and GC cell lines. mechanism - - -
NMRAL2P pseudogene/lncRNA 32469171 Cell proliferation assay; Flow cytometry; Transwell assay cell line - - - - - No Reports NMRAL2P overexpression induced a more malignant phenotype and significantly inhibited the expression of ACOT7. mechanism - - -
NMRAL2P pseudogene/lncRNA 32469171 Luciferase reporter assay cell line - - - - - No Reports NMRAL2P indirectly methylated ACOT7 by binding to DNMT3b, thereby suppressing ACOT7 expression. mechanism - - -
PCAT18 lncRNA 32119960 Cell proliferation assay; Cell invasion and migration assays; Cell colony forming assay; cell line - - - - - No Reports PCAT18 down-expression promoted proliferation, migration and invasion of gastric cancer cells. target - - -
PCAT18 lncRNA 32119960 Xenograft mice models mice - - - - - No Reports In in-vivo experiment, PCAT18 over-expression restrained tumor growth and metastasis. target - - -
PCAT18 lncRNA 32119960 Luciferase reporter assay; RIP cell line - - - - - No Reports PCAT18 is the direct target of miR-135b target - - -
SNHG7 lncRNA 29131253 qRT-PCR tissue China 68 68 Up - No Reports The qRT-PCR experiment showed that in a total of 68 cases of cancer tissues and tumor-adjacent tissues, the relative expression of SNHG7 was upregulated in 48 cases of gastric cancer tissues and 5 gastric cancer cell lines. target - - -
SNHG7 lncRNA 29131253 Cell proliferation assay; Flow cytometry cell line - - - - - No Reports The in vitro experiments showed that after SNHG7 expression was interfered, the proliferation of gastric cancer cells was inhibited with an increase in apoptotic rate and arrest of cell cycle in G1/G0 phase. target - - -
SNHG7 lncRNA 29131253 Xenograft mice models mice - - - - - No Reports Experiment on nude-mouse transplanted tumor model confirmed that after SNHG7 expression was interfered, in vivo tumor growth was inhibited. target - - -
UCA1 lncRNA 28569779 qRT-PCR tissue China 39 39 Up - No Reports UCA1 expressed highly in GC tissues and GC cells, overexpression of UCA1 indicated higher tumor stage and lymph node metastasis in GC patients. target - - -
  Summary of Functional Type.
Functional Type Count
diagnosis 5
prognosis 211
mechanism 143
target 360
target,diagnosis 1
target;prognosis 1
  Summary of Interaction.
Gene Interaction Gene PMID
ACOT7 NMRAL2P 32469171
ADAM10 miR-448 26852749
ADAM10 miR-320a 29152656
ADAM9 miR-126 28260063
ADAM9 miR-129-5p 29879625
AGAP2-AS1 CDKN1A; EZH2; LSD1; SP1; 28209205
AKT2 miR-143-3p 28404925
AKT2 miR-29a; miR-29b; miR-29c; 25428377
AKT3 miR-582-5p 29228422
ANKRD40CL miR-490-3p 32293550
ANXA4 miR-203 30837034
ASCL2 miR-223 30106147
ATF7 miR-103a-3p 29754469
BMI1 miR-15a 26894855
BRD4 CDKN1A 29434197
BTG2 miR-27a-3p 27409164
C8orf76 DUSP5P1 30733230
CBFB miR-204-5p 29386218
CCN1 miR-142-5p 30178386
CCND1 miR-9 23383271
CCND1 miR-155 26955820
CCND1 miR-193a-3p 29848678
CD151 ITGA3 23533596
CDC42 CDC42 25152372
CDH1 CDH1 30063010
CDK6 miR-1296-5p 30530570
CDK9 miR-613 28701053
CDKN1A BRD4 29434197
CLCN3 XRCC5 30217218
CLDN18 miR-1303 24647998
COL10A1 SOX9 30154451
COPS7A KRT19P3 31409899
CRMP1 miR-187 27864146
CRNDE MIR145 28490034
CTBP1 miR-644a 27983935
CTHRC1 let-7b 25510669
DDX5 miR-5590-3p 30029874
DNMT3A CDKN2C 26350239
DUSP5P1 C8orf76 30733230
EPHA8 ADAM10 31026069
FADD miR-675 28848149
GAS5 YBX1 25959498
hsa_circ_0000199 miR-198 30927924
hsa_circ_0000993 miR-214-5p 30215537
hsa_circ_0004771 miR-149-5p 30717751
hsa_circ_0092303 miR-331-3p 31182928
ITGA3 CD151 23533596
JAZF1 miR-1275 31357957
KDM4B CTNNB1 24077348
KRT19P3 COPS7A 31409899
LINC01234 Ago2; miR-204-5p; 29386218
MIR103A1 ATF7 29754469
MIR106B ARMCX1 30907988
MIR126 ADAM9 28260063
MIR1275 JAZF1 31357957
MIR1296 CDK6; EGFR; 30530570
MIR1303 CLDN18 24647998
MIR135B PCAT18; CLDN11; 32119960
MIR137 AKT2 26102366
MIR142 CYR61 30178386
MIR145 E2F3 28490034
MIR148A CCKBR 27518872
MIR152 CD274 28427226
MIR155 CCND1 26955820
MIR15A BMI1 26894855
MIR181A1 ATM 24531888
MIR187 CRMP1 27864146
MIR1915 BCL2 31036603
MIR193A ETS1; CCND1; 29848678
MIR194-1 SUFU 27810403
MIR198 hsa_circ_0000199; PIK3R1; 30927924
MIR199A1 TBL1XR1 30193607
MIR203A CASK 25373785
MIR203A ANXA4 30837034
MIR204 CBFB 29386218
MIR208A PDCD4 27634902
MIR208A SFRP1; MEG3; 29886152
MIR27A BTG2 27409164
MIR302B IGF-1R 28743112
MIR320A FOXM1; CDKN1B; 27086911
MIR331 circCACTIN; TGFBR1; 31182928
MIR335 Bcl-w; SP1; 21822301
MIR338 ZEB2 25945841
MIR338 ADAM17 26617808
MIR338 Rab14; Hhat; 30308487
MIR381 TMEM16A 28193228
MIR421 CASP3 25041019
MIR454 STAT3 30810117
MIR490 LINC00483; MAPK1; 32293550
MIR519D BCL6 29510377
MIR5590 DDX5 30029874
MIR582 AKT3 29228422
MIR589 LIFR; c-Jun; 30012200
MIR613 CDK9 28701053
MIR644A CTBP1 27983935
MIR675 H19 28848149
MIR874 AQP3 23800944
MIR9-1 CDX2 21225631
MIR9-1 CDX3 21225631
MIR9-1 CDX4 21225631
MIR9-1 CCND1; ETS1; 23383271
MIRLET7B AURKB 30237418
MIRLET7B CTHRC1 25510669
NMRAL2P ACOT7 32469171
PCAT18 miR-135b 32119960
PDCD4 miR-208a-3p 27634902
POU2F1 ECD 25678401
SFRP1 miR-208a 29886152
SIRT1 JUN 30250020
SNORD105B ALDOA 29554660
SOX9 COL10A1 30154451
STAT3 miR-454-3p 30810117
SUFU miRNA-194 27810403
XRCC5 CLC-3 30217218
YBX1 lncRNA GAS5 25959498

 Interaction graph The select node information.

  Summary of Hpylori.
Hpylori Type Count
Related 7
Not Related 3
No Reports 683
8
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